Facial palsy (Bell’s Palsy)

The CMGs are guidelines on the diagnosis and management of a range of common and rare, but important, eye conditions that present with varying frequency in primary and first contact care.

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Facial Palsy

 

 

 

 

 

 

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Aetiology

Paralysis of facial nerve (VII cranial nerve)
This Clinical Management Guideline addresses Bell’s Palsy (idiopathic lower motor neurone facial nerve dysfunction), which constitutes 72% of all facial palsy:

  • annual incidence 20-30 per 100,000, especially between 15 & 45 yrs with a possible second peak in the over-70s
  • sudden onset, unilateral
  • M = F (except in pregnancy, see below)
  • cause unknown, but sometimes associated with latent virus infection (HSV type 1, Herpes zoster)
  • fair prognosis without treatment 
    • however, up to 30% of treated patients do not recover completely
  • better prognosis with systemic steroid therapy (with or without anti-viral)
  • most improvement occurs within three weeks

Other causes of paralysis of the facial nerve, which are not addressed in this Clinical Management Guideline, include:

  • infection, e.g. otitis media
  • trauma, e.g. temporal bone fracture
  • tumour compressing the facial nerve, e.g. acoustic neuroma
  • sarcoidosis
  • Guillain-Barré syndrome
  • cerebrovascular accident (stroke)

Predisposing factors

Bell’s Palsy is more common in:

  • pregnancy (annual incidence increases to 45 per 100,000)
  • diabetes
  • HIV

Symptoms

Distressing cosmetic change due to loss of muscle tone on one side of face
Watering of eye (epiphora)
Ocular exposure causes:

  • redness, discomfort, pain, photophobia, reduced vision

 

Signs

Unilateral facial weakness including orbicularis oculi

  • incomplete blink leads to corneal drying
  • incomplete closure at night (lagophthalmos) causes prolonged corneal exposure
  • loss of lacrimal pump mechanism produces pooling and epiphora

Conjunctival hyperaemia, oedema, staining
Corneal desiccation ranges from mild superficial punctate erosions to frank ulceration (usually inferior)

Differential diagnosis

Other causes of facial nerve palsy:

  • as part of a stroke (cerebro-vascular accident with hemiplegia), in which forehead muscle function is preserved
  • infection (e.g. otitis media, Lyme disease)
  • trauma (e.g. cranial fracture, facial laceration)
  • tumour (e.g. acoustic neuroma: damage to nerve by tumour or secondary to surgical trauma; also perineural invasion by a squamous cell carcinoma)

Ectropion or Entropion
Other causes of lagophthalmos

  • orbital (thyroid eye disease – assess by exophthalmometry)
  • mechanical (cicatricial – look for lid scarring)
  • physiological (patient’s partner to check for full lid closure at night) 

Management by optometrist

Practitioners should recognise their limitations and where necessary seek further advice or refer the patient elsewhere

GRADE* Level of evidence and strength of recommendation always relates to the statement(s) immediately above

Non pharmacological

Tape lids closed at night (and during day if corneal desiccation is marked)
(GRADE*: Level of evidence=low, Strength of recommendation=strong)

Sunglasses for photophobia and general protection
(GRADE*: Level of evidence=low, Strength of recommendation=strong)

Therapeutic contact lens (if unresponsive to frequent use of ocular lubricants)

  • silicone hydrogel should be considered as first choice (however, scleral lens gives maximum protection)

NB therapeutic contact lens fitting is contraindicated in cases of neurotrophic keratitis with loss of corneal sensation (cranial nerve V). Such patents are at high risk of infection, which may be further increased by contact lens wear
(GRADE*: Level of evidence=low, Strength of recommendation=weak)

 

Pharmacological

Tear supplements / lubricants by day, unmedicated ointment at night
(GRADE*: Level of evidence=low, Strength of recommendation=strong)

Management category

New cases, and where there is loss of corneal sensation:
A2: first aid measures and emergency (same day) referral to GP or ophthalmologist
Improved prognosis in moderate/severe cases of Bell’s palsy if treated with systemic corticosteroid (with or without anti-viral) within 72 hours of onset
NB: corneal ulceration due to exposure is potentially sight threatening and may justify emergency referral

Recovering and established cases:
B2: alleviation/palliation; no referral

If cannot be managed easily, then:
B1: prescription of drugs; routine referral

Possible management by ophthalmologist

Urgent medical treatment for new cases:

  • systemic steroid ± anti-viral

Temporary tarsorrhaphy

Upper lid lowering with botulinum toxin injection of levator muscle

Surgery for permanently unrecovered cases:

  • tarsorrhaphy (permanent)
  • upper lid lowering (surgery, gold weight)
  • surgical sling to raise lower lid

Evidence base

*GRADE: Grading of Recommendations Assessment, Development and Evaluation (www.gradeworkinggroup.org)
 

Sources of evidence

Baugh R, Basura G, Ishii L, et al. Clinical practice guideline: Bell’s palsy. Otolaryngol Head Neck Surg. 2013;149(3)(suppl):S1-S27

de Almeida JR, Guyatt GH, Sud S, Dorion J, Hill MD, Kolber MR, Lea J, Reg SL, Somogyi BK, Westerberg BD, White C, Chen JM; Bell Palsy Working Group, Canadian Society of Otolaryngology. Management of Bell Palsy: clinical practice guideline. Head and Neck Surgery and Canadian Neurological Sciences Federation. CMAJ. 2014;186(12):917- 22

Madhok VB, Gagyor I, Daly F, Somasundara D, Sullivan M, Gammie F, Sullivan F. Corticosteroids for Bell's palsy (idiopathic facial paralysis). Cochrane Database Syst Rev. 2016;7:CD001942

Gagyor I, Madhok VB, Daly F, Somasundara D, Sullivan M, Gammie F, Sullivan F. Antiviral treatment for Bell's palsy (idiopathic facial paralysis). Cochrane Database Syst Rev. 2015;11:CD001869

Lay summary

Facial palsy results if the nerve supplying the muscles of the face, including the circular muscle around the eye, stops functioning. There are many causes, but Bell’s Palsy is the commonest, accounting for nearly three quarters of all cases. Usually this affects only one side of the face and is temporary, lasting around three weeks, though recovery may not be complete. The cause is unknown. People between the ages of 15 and 45 are most likely to be affected, but the condition is commoner in those who are pregnant, have diabetes or are living with HIV infection.

Patients notice that the affected side of the face droops and does not move. The eye may not close properly and as a result it can become red, uncomfortable and watery. The optometrist will examine the eye for signs of drying and for loss of feeling, which sometimes occurs. New cases will be referred as emergencies to the GP or the ophthalmologist, as recovery is improved if steroid tablets are given within 72 hours of the onset of symptoms. Longer-standing cases are managed by the optometrist and if necessary referred routinely to the ophthalmologist.

The optometrist will usually prescribe artificial tears to be used frequently during the day and ointment at night. Taping the eyelids closed at night may help. Sunglasses will often relieve light sensitivity and physically protect the eye. Sometimes a contact lens will be fitted to protect the cornea (the clear window of the eye).

Facial Palsy (Bell’s Palsy)
Version 12
Date of search 20.11.17
Date of revision 29.03.18
Date of publication 09.05.18
Date for review 19.11.19
© College of Optometrists

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